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Conversely, cartilage-specific overexpression of GPX4 via transgenic technology, or restoration of its activity using small molecule drugs such as certain natural compounds or ferroptosis inhibitors, can significantly delay cartilage degeneration, improve subchondral bone sclerosis and osteophyte formation, and alleviate synovitis in model animals (Xiao et al., 2024), which directly confirms the protective role of GPX4 in OA progression from a gain-of-function perspective

doi: 10.1021/acs.jafc.8b07117 44 YuX.-J.WangZ.-P.LiangM.-J.WangZ.LiuX.-Y.HuL.et al

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References Gamella M et al (2014) Activation of a biocatalytic electrode by removing glucose oxidase from the surfaceapplication to signal triggered drug release